Investigating MND for all contributing causes.

Foreword *

The purpose of this paper is – as the name implies – to collate all the known causes and complications of MND – both direct and indirect – as well as treatments for the purpose of attempting to perhaps initially stabilise the disease progression as the first step towards the disease.

Then hopefully, eventually reversing the outcome if enough nerve cell and muscle repair can occur, the known innate ability of neuroplasticity may enable, eventually, a return of at least some nerve cell activity.

And, given the current lack of known treatments, to use a different treatment approach once the known or suspected causes have been determined would be to use in combination the various treatments known to be effective against the known or suspected causes, as listed below.

The justification for this “connect the the individual silos of knowledge into a concerted attack” approach is that the current, more allopathic approach, has failed, the treatments are harmless and – as the late Richard Scolyer has said – why not “give it a crack”.

Introduction

While herpes family viridae have been dismissed as neurotoxic agents that cause cell death in adults, the largest member of that family – cytomegalovirus – along with others has been implicated in the affecting of normal responses from immune cells called T cells in behaving differently from the other cells of that family, in being able to divert antibodies from attacking the viral surface to that of the inner neuronal structures that it mimics. It basically produces “false flag” proteins to draw attack from the TH1 immune system away from the virus itself, and so depleting its attack by exhausting its activity.

Below quotes directly a report from the National Institute of Health to avoid an unintended misquoting in the attempted summary above.

The gradual onset of necrosis and cell death¹ of neuronal material within nerve cells by specific herpes virion also cannot be discounted as possibly happening in “rare cases” much as happens in embryos. And given the harmless nature of using a combination natural therapies, and with the patient’s consent, it is my belief that that approach is both ethical and viable.

Rarity of cases.

Therefore, if adult MND is such a rare (and incurable) condition, then it is reasonable to hypothesise that in rare case of infants acquiring cell death from CMV infection that it is also possible, though rare, for intracellular cell death in an adult in 1 in 100,000 cases. And if in infants then why not in adults in rare cases?

In other words, because orthodox measures have failed to curb this disease, unorthodox measures of poly-treatment, poly-system approach – yet harmless to the patient – must be considered as a viable alternative.

Nerve myelin sheath and demyelination

Brain and motor nerves are coated with a fatty substance called myelin whose purpose is to insulate the nerves from their surroundings and to prevent the nerve from “shorting out” into the surrounding fluids and structures. Herpes viridae have the effect of eroding myelin, reducing the nerves’ ability to function. This loss of function can result in fatigue and loss of motor activity.

Known or suspected direct or indirect complications/contributors of MND and suggested treatments – summary

1) Cytomegalovirus

2) “junk” DNA and dormant “ancient” viridae.

3) Known oncogene and tumour suppressor gene stabilisers

4) Chronic traumatic encephalopathy

5) Obstructive sleep apnoea

6) Known  antioxidant deficiency

7) Immune challenge

8) Virus-propagating dietary elements to avoid e.g arginine foods

9)  Dietary  deficiency of inhibitors² of viral reproduction e.g. tyrosine foods, lysine and “activation” of grains

10) Microbial infections and parasites

11) Insufficient soaking of foods containing phytic and oxalic acids before consumption

12) Consumption of pesticide-containing foods

13) Microbial, yeast and parasitic infections that may invade immune-challenged nerve (and other) tissue

Natural inhibitors of above challenges

1) Herbal and pharmaceutical inhibitors of herpes virus – especially CMV.

2) Myelin-building nutrients

3) Neurone supporting herbs, nutrients and meds

4) Herbal stimulants of immune activity

5) Vitamin/nutrient/dietary stimulants of immune activity

6) Nerve-specific anti-inflammatories – herbal, nutritional and/or pharmaceutical

7) Nutritional elements supporting nerve axonal or dendritic membrane and muscle growth

8) Known nutritionals that support neuronal ATP production of energy and other functions

Treatments that may affect nerve and muscle structural integrity

Frequency Specific Microcurrent to frequencies that may support pain and inflammation, ATP and Endorphin production, reduce cellular cytokines, reduce tissue necrosis, repair tissue and inhibit virus activity and emerging pathology where identified.

 

Differential diagnosis	Suggested Treatments
CMV	Sod Ascorbate, lysine; FSM; anti-viral med; dietary deletions e.g. arginine; dietary additions eg tyrosine; delete pesticides; have organic foods; soak oxalates and phytates:
Junk DNA “ancient virus”	Cell nutrient; above
DNA attack by herpes viridae	Curcuminoids; Hydrolysed citrus pectin; above
Immune compromise	Above; ginkgo; Essiac; Phil’ssmoothi
Intra neurone bacterial infections; gut dysbiosis;	Probiotics – acidophilus, bifidus, rhamnosis, paracacea, plantarum
Parasites	Mebendazole, vlack walnut; a Artemisia annua, absinthium.probiotics; whey, colostrum
Brain neurone decline	Bacopa, ginkgo, lecithin, above
Undernutrition	Smoothi
Atp support	Ubiquinol + FSM
Brain hypoxia	CPAP
All above causes	FSM
Sinus infection	Saline wash; probiotics; gaviscon; Losec;
Refluix	Surgery on disphragm plus abover
OSA	CPAP plus above

* Given the lack of medical treatment available, it will be agreed that if suggested treatments/meds are harmless and beneficial, then they shall be approved, pending the consent of the patient. It will be agreed that the various meds and nutrients will be compatible and compounded in as few units as possible in as least invasive manner as possible that is tolerable to taste.

Any medical anti-inflammatory drugs will be non-steroidal in nature of necessity so as to maximise immune potential, given the immune-compromising effect of that category.